Correct! If we could control the telomerase to help lengthen our telomeres, we could potentially slow down age-related cellular decline and get a few more years out of our lives. Maybe there should be equal effort put on the research of telomerase as well as the most common diseases and complications that are currently affecting us.
They have proven that you could use Epitalon peptide to lengthen telomeres. So there you go, problem solved. 🤣 Question: Why do our body have telomerase enzyme to extend telomeres if it won't use it ? Obviously there is something controlling the production of telomerase enzyme, instead of keep yapping about how telomeres getting shorter people should talk about what controlling the telomerase enzyme.
I agree - if we can control the telomerase effectively then it is worth the effort and research. To answer your question, the body does use the telomerase enzyme but not frequently in somatic cells. It uses it in germ cells, and stem cells, that divide frequently to maintain chromosomal stability. In somatic cells however, the body sometimes can sometimes "get it wrong". When there is a "bad" cell, the body will take it out with apoptosis. Sometimes the body misses these bad cells, and this can happen more often than we think. Some of these cells then turn cancerous. And the older cells generally are, the more likely they are to have DNA damage, or other damage that could become cancerous. If we keep lengthening these somatic cells lives indefinitely, they will eventually all become cancerous. Instead, maybe we should work out ways we can regenerate our bodies from stem cells as a way to length our organisms life as a whole, as when we create new cells from stem cells, they will not be aged or damaged.
@@teachyaa true, but we wont be able to regenerate a brain so yet again the main goal comes down to how will cancer be delt with? With how much money goes into cancer each year will there ever be a cure? Is there perhaps a Phoebus cartel in the medical world stopping all cancer research? whole lotta questions and I ask them because Id like to live forever.
@@Daddy_is_here12321 Cells generally undergo several mutations-typically around seven-before they are classified as cancerous. The body has mechanisms, like apoptosis, to detect and eliminate mutated cells. However, some mutated cells evade detection and survive long enough to become cancerous. Preventing cancer is challenging because it can arise from various causes, such as errors during cell replication or exposure to gamma rays from the sun. Mutated cells become problematic when they start behaving independently of normal cellular controls. One of the biggest challenges with cancer is not necessarily the lack of treatments but rather the difficulty in detecting it early enough. Early detection and prevention are crucial because once cancer spreads extensively, it can become much harder to treat effectively. There has been extensive research into different types of cancer and treatments. However, each type of cancer "appears" differently-some mimic healthy cells, making them difficult to target. While significant advancements in cancer research are made each year, the diversity of cancer types and their unique mutations make a universal cure unlikely.
not if it means you just get super frickin decrepit or trapped in a fate worse than death
What if you just become a robot
So it should be possible in theory, but in practice you would die of diseases or complications.
Correct! If we could control the telomerase to help lengthen our telomeres, we could potentially slow down age-related cellular decline and get a few more years out of our lives. Maybe there should be equal effort put on the research of telomerase as well as the most common diseases and complications that are currently affecting us.
Wouldn't want to
the way things are at least...
They have proven that you could use Epitalon peptide to lengthen telomeres. So there you go, problem solved. 🤣
Question: Why do our body have telomerase enzyme to extend telomeres if it won't use it ? Obviously there is something controlling the production of telomerase enzyme, instead of keep yapping about how telomeres getting shorter people should talk about what controlling the telomerase enzyme.
I agree - if we can control the telomerase effectively then it is worth the effort and research.
To answer your question, the body does use the telomerase enzyme but not frequently in somatic cells. It uses it in germ cells, and stem cells, that divide frequently to maintain chromosomal stability. In somatic cells however, the body sometimes can sometimes "get it wrong". When there is a "bad" cell, the body will take it out with apoptosis. Sometimes the body misses these bad cells, and this can happen more often than we think. Some of these cells then turn cancerous. And the older cells generally are, the more likely they are to have DNA damage, or other damage that could become cancerous. If we keep lengthening these somatic cells lives indefinitely, they will eventually all become cancerous.
Instead, maybe we should work out ways we can regenerate our bodies from stem cells as a way to length our organisms life as a whole, as when we create new cells from stem cells, they will not be aged or damaged.
@@teachyaa true, but we wont be able to regenerate a brain so yet again the main goal comes down to how will cancer be delt with? With how much money goes into cancer each year will there ever be a cure? Is there perhaps a Phoebus cartel in the medical world stopping all cancer research? whole lotta questions and I ask them because Id like to live forever.
@@Daddy_is_here12321 Cells generally undergo several mutations-typically around seven-before they are classified as cancerous. The body has mechanisms, like apoptosis, to detect and eliminate mutated cells. However, some mutated cells evade detection and survive long enough to become cancerous. Preventing cancer is challenging because it can arise from various causes, such as errors during cell replication or exposure to gamma rays from the sun. Mutated cells become problematic when they start behaving independently of normal cellular controls.
One of the biggest challenges with cancer is not necessarily the lack of treatments but rather the difficulty in detecting it early enough. Early detection and prevention are crucial because once cancer spreads extensively, it can become much harder to treat effectively.
There has been extensive research into different types of cancer and treatments. However, each type of cancer "appears" differently-some mimic healthy cells, making them difficult to target. While significant advancements in cancer research are made each year, the diversity of cancer types and their unique mutations make a universal cure unlikely.