Dr. Gobind Rai Garg discusses the topic - Recent advances in Migraine
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- เผยแพร่เมื่อ 5 ก.พ. 2025
- Watch Pharmacology Guru, Dr. Gobind Rai Garg discuss the topic - Recent advances in Migraine. For more such videos by India's best Pharmacology faculty, Dr. Gobind Rai Garg download the app -
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U r really a great techer... Thanks a lot sir👍😊
My pleasure dear and thanks a lot for nice words
All formulations not available in india
Sir, is Sumatriptan safe in pregnancy and lactation???
Nice sir .why inflammation of blood vessels in brain
Cgrp is released which acts on meningeal blood vessels and cause vasocnstriction
Go for ranjan kumar hes the best
Why you are wasting your time here dear
@@DrGobindRaiGarg i m not wasting my time sir i just told my view
Good to know
Where can I got full all pharmacology lecture in sema Hindi
Wow💕 lots of love and respect from Pakistan 🇵🇰
Thanks dear
thaks sir
sir monoclonal antidody against CGRP is ERTUNUMAB or ERENUMAB ? on Google ERENUMAB is given .
which is more correct ?
Erenumab is correct
Dr. Gobind Rai Garg Thanks sir
Thank u sir
Sir please explain how calcium channel blockers and anti depressants used for migraine prophylaxis (MOA)
These Inhibit release of CGRP
Thank q sir
You Re good!
Thanks a lot dear
Nice sir realy good explosion
Thanks dear
Sir pls tell what is the mechanism of action of Propranol in prophylaxis of migraine
Simply prevent beta 2 mediated vasodilation
Thanks sir..but I am confused that Propranol causes vasodilation for treatment of hypertension... And in case of migraine it causes vasoconstriction. So do it acts by different mechanism in both situations?
May b due to the membrane stabilization activity.
The drugs used in prophylaxis are actually vasodilators since the trigger event is followed by ischaemia...only later on there is vasodilation and pain.
Drugs in prophylaxis are dilators to prevent ischaemia whereas drugs in acute management are constrictors.
Beta blockers reduce the cardiac output and in the long run the body adapts to reduced cardiac output by reducing the peripheral resistance. That's how it acts as antihypertensive. Also it reduces renin release.
Hope this clears your doubt..
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Sir why methysergide a 5-HT antagonist is used in prophylaxis of migraine
It is an ergot derivative and possess direct to cause smooth muscle contraction
One of the theories of migraine suggests that people with migraine have a v low level of serotonin. Their brain is adapted to these low levels. But during stress or following the trigger event.. the serotonin levels rise. body reacts to this high level of serotonin with pain.
Methysergide is a serotonin antagonist and thus effective. Hope this clears your doubt 🙏🏼
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Sir. That z ERENUMAB CGRP antagonist , with the reference of KDT
Yes, please correct
Super explanation sir....
Thanks dear
♥️
Dear dr gobind garg sahab can i contact you
Sure. Please mail me at gobind_garg@yahoo.co.in
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