วีดีโอ

Thanks you so much

I have a question Professor. The interaction of protein K and Y before the sumoylation is unstable and Sumoylation bring the protein interactin a long stable term interaction between K and Y? Thanks for your video, it's really well explained and helpful for my MSc degree. Best Oscar

Invasion of privacy

Thank you for such a detailed video. Just out of curiosity, are you writing backwards on some transparent material?

Very helpful video. Very well explained with examples and live demonstrations. Thank you❤

best ever explanation

Hello, dear Dr. Differently this video helpful THANK YOU

❤

Great explanation

Omg no one is doing mirrors 🪞 that’s toooo much

So how can I prepare crystal violet to use it in this assay

What about renoving the neutralized trypsin and adding fresh media ?

This helped me alot! Thank you maam ! ❤ may you live a long and meaningful life ❤❤❤😊

you just saved my life

clear explanation indeed .

what the cringe I just watch

thank you doctor, good expanation!

I think the answer to the question will be 25% in Pre-G1 (Apoptotic), 25% in S phase, and 50% in G2/M phases. Because, during the treatment time, the are two equivalent time-periods for the cell death. If the total cell cycle period lasts 24 hours, cells that have at least passed 18th hour and had only 6 hours left to finish the cell cycle will undergo apoptosis (which corresponds to quarter of all cells). Within 12 hours, all G1 phase cells will be at S or G2-M phases, which substitutes previous S phase cells and their remain as a quarter. Another quarter goes to G2-M phase coming from the G1 phase. Lastly, the remaining 25% will also be in G2-M phase (particularly in M phase) due to arrest which came from the 12-18th hours in the beginning of the experiment but did not die.

Bilkent - Cell Biology II - salutes to Onur Hoca :)

really helped in my campbell biology exercise

Learning question about E2F1 and 7/8. If E2F is getting activated via PI3K-Akt-Signaling (due to contact with ionized LNP) and you see E2F8 1.5 fold up but E2F1 not supressed: Should this happen? As far as I understood: E2F8 acts as supressor of E2F1 especially when triggered by Akt-pathway?

Awesome 👌

Thank you Doctor Rosas for your immense contribution to explaining flowcytometric analysis 👍

Thanks for the information

👍

well prepared and clear

A̺m̺a̺z̺i̺n̺g̺ v̺i̺d̺e̺o̺ ❤

Thank you <3

Thank you so much 🙏🙏🙏❤❤❤❤❤❤❤❤❤❤

Thank you very much for this teaching..

Many thanks for your information. I'm curious about how can we record the video cell proliferation process in the incubator?

Thanks sir for such beautiful explanation!👍

Such an underrated video

Thanks!!!!!!

How many cells per well if we use a 96 plate? 72h treatment... I know it varies, but my cells grow a lot, I wonder what you would suggest haha

Great idea, Please keep on this good work.

THANK YOUUU this is so clear and helpful

Maam , during gram staining , accidentally Cristal violet fall on my hand , I am searching Google about Cristal violet , it show me Cristal violet has carcinogenic and mutagenic ability .Now I am scared can you tell me I am in risk or not 😢

This is the video I needed to see! Thank you Dr Rosas!

I think that the answer is: 25% apoptotic, 8.3% G1/G0, 25% S, and 41.7% G2/M

How can we count the cell?? In cell cycle

This is barely high school level. It would be great to have something for uni students

Thank you Dr. Rosas Acosta! 😳

Great teacher

Thank you for making this video! it helps a lot! great quality:)

Thank you dr Roase The video is very helpful ❤

Fantastic video, you’re a gifted speaker

But DMSO in cell culture causes cytotoxicity in comparison to cells with no DMSO or treatment as a control too.

Promo SM