I am extremely critical when it comes to the methodology of executing knowledge from a donor like you to others like spectators since I employ anthropological tools and I tell you with ease that you accomplished very well in your clip and thus I was compelled to subscribe! 😊
I think with isolated right side hf, u shouldn’t treat it with diuretics cause the systemic bp is already low due to decreased preload of the left ventricle. Instead, inotropes will help the right ventricle to pump more blood and regain the circulation and blood pressure.
Should mention the role of atrial fibrillation in worsening heart failure or causing heart failure in some cases Heart that produces adequate blood flow at rest but only when filling pressure is somewhat elevated can be described as having "mild" heart failure? (Not functioning on the proper part of the Starling curve) New atrial fibrillation in a heart with previous mild left ventricular hypertrophy could cause reduced end diastolic volume in the left ventricle causing a form of diastolic heart failure ?
Thank you for that great video, I would appreciate your answer for this question : in case of increased heart beat in a normal person during exercises, (as heart pumps harder) would that in any way cause enough hypertrophy to the left ventricle that would result in systolic or diastolic heart failure? Thanks
In the case of exercise, the heart muscles do increase in size however, there is no pathological consequence if the person is healthy. In fact, the muscles become even more efficient at pumping blood with prolonged cardiovascular exercise, which is the reason why endurance athletes have lower resting heart rates - It takes less effort for the heart to pump enough blood around the body. I think research is currently being done to find out specifically why hypertrophy is good in exercise but bad in heart disease.
Exercise induced hypertrophy doesn't cause heart failure. Because in that case, hypertrophy happens in eccentric manner. So it doesn't result in decrease of ventricular volume.
Depending on type of exercise and whether sustained at a high metabolic level for a long time there could be both some hypertrophy and some volume increase "dilation" of the heart chambers It is suspected that there is a heightened risk of the appearance of atrial fibrillation in older people who have done such as winning 100 mile footraces or professional bicycle races,perhaps cross country ski races while they were in the prime years of life
There are now methods available that allow more careful assessment of the bronchial vascular bed non-invasively in humans. Soluble gas techniques combined with high resolution X-ray CT scanning will allow for the assessment of the interplay between functional bronchial blood flow and its effects on airway structure and pulmonary function valuable tool to determine this circulation’s effect in other pathologies such as HF.
How so? An example being - You have a I.V. drug user who develops endocarditis and vegitation on the tricuspid valve. If left untreated this eventually progresses to a state that causes right sided HF and may eventually progress to biventricular HF. So right sided HF can cause left sided HF. (EDIT: GRAMMAR)
@@mariusrulz Its not an uncommon thing among drug users and you see it somewhat often in the cardiovascular field. You may also also see PHT cause right sided HF and that leads to left sided HF. This is just a quick reply and can easily link you sourced material from google scholar after work.
So, for weeks I’ve been getting worsening chest pressure/dullness. Which is becoming more frequent, and also more seemingly severe each time. Finally went to hospital yesterday on when it was extremely severe and I was dizzy etc as well. ECG was fine. Triponin or whatever was a “2” and they would have been worried for me if it was 5 or more. The problem is, I’m a 25 year old fit looking male. I work out. But I also smoke a pack a day and eat the absolute worse shit. I don’t believe I’m an anxious person. But it almost feels like this crushing pressure is being entirely ignored by doctors who seem to all but be acting like “you’re an idiot, you’re too young and fit to be worried about your heart”. Also, during these periods, my blood pressure is always waaay higher than usual. 170/99 yesterday at the hospital, whereas I regularly sit at about 120/70ish. They told me it was just because I was stressed (I replied “i don’t feel stressed. Work is good. Mrs is good. Money is good - I’m not even scared about this I just want to know what is going on, because obviously something is). This spread to my back once about 4 days ago. Lasts from 30 mins to 1.5 hours. Like idk what to do. Doctors said I could have more tests done, but gave me a hand out saying (in short) “of people presenting like you, only 1 in 100 has a confirmed heart attack within 30 days). And said I could sign a form to be released, or make them do more tests. I got released because they were honestly basically talking down to me and making assumptions about my mental state etc. So anybody experienced something like that? Strong crushing pressure right in heart area, increasing from once every 3 or 4 days to every 2 days to about every day? Increasing in intensity? Anyone end up finding out what it was?
you should really stop smoking completely n have a healthy diet for avoiding further complications. a pack a day is sooo unhealthy and you dnt even know what you are doing to ur body.... this is ridiculous.
This systematic review and meta-analysis identified significant associations of both circulating and dietary magnesium and risk of CVD events. Circulating magnesium (per 0.2-mmol/L increment) was associated with a 30% lower risk of CVD, with trends toward a lower risk of IHD and fatal IHD. Dietary magnesium was associated with a 22% lower risk of IHD and showed a nonlinear association with fatal IHD, with a 27% lower risk up to a threshold of ∼250 mg/d, compared with lower intakes. This investigation, which included a total of 313,041 individuals in whom 4106 CVD, 3215 IHD, and 1528 fatal IHD events were documented for circulating magnesium and 7889 CVD, 4319 IHD, and 1158 fatal IHD events for dietary magnesium, provides the most robust evidence to date of the associations between circulating and dietary magnesium across their usual physiologic ranges and CVD risk.
You’ve been a lifesaver osmosis!! Your contributions to our knowledge is indescribable!! Much appreciated
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May God forever bless Osmosis. You literally saving all med students 🙏🏽💖😭
Osmosis is just love!! Life saving !!! Most amazing videos i have ever seen on youtube for medical studies! What would i do without you osmosis!❤💕💕
We still live in the midle ages
Incredible
One of the best tutorial videos I have watched. Thank you!
I think 10:39 is wrong, shunt causes eccentric hypertrophy by volume overload and not concentric
This is probably the best explained video I have found on medical information. Thank you! ❤️
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Don’t know how to thank Osmosis after i get my medical degree ! 😊
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Really good video for quick recap of pathophysiology. I wish you would go more in depth with the treatment in your videos though, it would help a lot.
Loved how they even added the pulmonary edema sound
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Osmosis is on passmedicine and also medscape? Well done!! You've helped us alot in our medical journey
I am extremely critical when it comes to the methodology of executing knowledge from a donor like you to others like spectators since I employ anthropological tools and I tell you with ease that you accomplished very well in your clip and thus I was compelled to subscribe! 😊
Thank you so much, verywell explained. I hv a doubt though what happens to afterload?
Absolutely fantastic video, very well explained!
5:04, so if stent is placed, the scar produced on heart muscle will cure on its own?
Thank you so so much. This has finally made sense today after watching your video.
Great help while studying Pharmacology.
perfect explanation , you are the best OSMOSIS , thank you ❤❤❤
This is really good
I think with isolated right side hf, u shouldn’t treat it with diuretics cause the systemic bp is already low due to decreased preload of the left ventricle. Instead, inotropes will help the right ventricle to pump more blood and regain the circulation and blood pressure.
The explanation was just perfect
One time in the future people won't need to go to a university coz everything will be available in TH-cam insha allah
Amazing explanation ❤👏 good job osmosis
Should mention the role of atrial fibrillation in worsening heart failure or causing heart failure in some cases
Heart that produces adequate blood flow at rest but only when filling pressure is somewhat elevated can be described as having "mild" heart failure? (Not functioning on the proper part of the Starling curve)
New atrial fibrillation in a heart with previous mild left ventricular hypertrophy could cause reduced end diastolic volume in the left ventricle causing a form of diastolic heart failure ?
Perfect! Just perfect! Thank you so much for this amazing video!
Excellent video
this is absolutely amazing demonstration good luck
Life saving concepts 💞
after searching tons of websites, finally found here, what i was looking for
thnkw
It was awesome. I really appreciate your work.
please can anyone answer me ? the ascite's liquide is transudate or exudate ??
we need to calculate with lightes criteria
Thanks a lot dude, you guys saved my ass of this exams ♥️♥️
Mines too😂
Thank you for that great video, I would appreciate your answer for this question : in case of increased heart beat in a normal person during exercises, (as heart pumps harder) would that in any way cause enough hypertrophy to the left ventricle that would result in systolic or diastolic heart failure? Thanks
In the case of exercise, the heart muscles do increase in size however, there is no pathological consequence if the person is healthy. In fact, the muscles become even more efficient at pumping blood with prolonged cardiovascular exercise, which is the reason why endurance athletes have lower resting heart rates - It takes less effort for the heart to pump enough blood around the body. I think research is currently being done to find out specifically why hypertrophy is good in exercise but bad in heart disease.
@@amz_mansaray Thank you.
Exercise induced hypertrophy doesn't cause heart failure. Because in that case, hypertrophy happens in eccentric manner. So it doesn't result in decrease of ventricular volume.
Depending on type of exercise and whether sustained at a high metabolic level for a long time there could be both some hypertrophy and some volume increase "dilation" of the heart chambers
It is suspected that there is a heightened risk of the appearance of atrial fibrillation in older people who have done such as winning 100 mile footraces or professional bicycle races,perhaps cross country ski races while they were in the prime years of life
Absolutely fantastic but just a bit speed s fast...
Thank you alot ❤️ I really enjoyed this video ❤️💕
U guys r love thankkkk uuuuuu jazakillah khairan❣️
Can you please make video on arrythmia !!
Are they changed their channels name? This is *osmosis* right?
Great video like always osmosis
we need to calculate the flyid content
Magnificent video ...thanks alot ♥️
Really great video helps alot
Best Video ever 🔥
Very well explained
Thankyou
Thank you so much for this video
perfect explanation
great video!
If ventricles contract harder, hypertrophy would occur. So their should be thinning of walls in dilated cardiomyopathy or am I missing something
Nice explanation 👍
Osmosis is awesome!
Can ı ask a question? what is the diffirence between arrhythmia and dysrhythmia in terminology? thanks.
I think the word "dysrhythmia" is more likely to be used when describing abnormal tracings in the EEG (electroencephalogram) "brain waves"
perfectly explained thank u!
Thank you! 💖
thanks. it was well explained
thank you
Please can u till me how u make or produce such video
very productive video
I don't know how to thank you it's great it's amazing it's nice it's fantastic thanks a lot 😍
Fantastic video!
Best video. Simplified and covered every aspect of HF Subscribed nd liked
Very nice video 🤗🤗
Entire Harrison knowledge in a span of 15 min
Thank you💕
Helpful video
great information
Amazingly concrete video. Many thanks!
Tq u so much,this is just awesome
Really good video, thank you ! :)
There are now methods available that allow more careful assessment of the bronchial vascular bed non-invasively in humans. Soluble gas techniques combined with high resolution X-ray CT scanning will allow for the assessment of the interplay between functional bronchial blood flow and its effects on airway structure and pulmonary function valuable tool to determine this circulation’s effect in other pathologies such as HF.
Such a great video but i need indonesian translate please... Thankyou for uploading this ❤
Thank you endlessly ❤️
Excellent
Nice skill
4:25 - Left sided heart failure can cause right sided heart , but vice versa is NOT true . Thats wrong . Please correct it
How so? An example being - You have a I.V. drug user who develops endocarditis and vegitation on the tricuspid valve. If left untreated this eventually progresses to a state that causes right sided HF and may eventually progress to biventricular HF. So right sided HF can cause left sided HF. (EDIT: GRAMMAR)
Add a reference to a standard textbook where it is mentioned so. I haven't found any yet.
@@sacordle Add a reference to a standard textbook where it is mentioned so. I haven't found any yet.
@@mariusrulz Its not an uncommon thing among drug users and you see it somewhat often in the cardiovascular field. You may also also see PHT cause right sided HF and that leads to left sided HF. This is just a quick reply and can easily link you sourced material from google scholar after work.
Thank you 😙😙😙 osmosis
Thank you ☺☺☺
thank youuuuuuu
amazing thank u
Great video !! Subscribed √
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Thanks.
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So, for weeks I’ve been getting worsening chest pressure/dullness. Which is becoming more frequent, and also more seemingly severe each time.
Finally went to hospital yesterday on when it was extremely severe and I was dizzy etc as well. ECG was fine. Triponin or whatever was a “2” and they would have been worried for me if it was 5 or more.
The problem is, I’m a 25 year old fit looking male. I work out. But I also smoke a pack a day and eat the absolute worse shit.
I don’t believe I’m an anxious person. But it almost feels like this crushing pressure is being entirely ignored by doctors who seem to all but be acting like “you’re an idiot, you’re too young and fit to be worried about your heart”.
Also, during these periods, my blood pressure is always waaay higher than usual. 170/99 yesterday at the hospital, whereas I regularly sit at about 120/70ish. They told me it was just because I was stressed (I replied “i don’t feel stressed. Work is good. Mrs is good. Money is good - I’m not even scared about this I just want to know what is going on, because obviously something is).
This spread to my back once about 4 days ago. Lasts from 30 mins to 1.5 hours.
Like idk what to do. Doctors said I could have more tests done, but gave me a hand out saying (in short) “of people presenting like you, only 1 in 100 has a confirmed heart attack within 30 days). And said I could sign a form to be released, or make them do more tests. I got released because they were honestly basically talking down to me and making assumptions about my mental state etc.
So anybody experienced something like that? Strong crushing pressure right in heart area, increasing from once every 3 or 4 days to every 2 days to about every day? Increasing in intensity? Anyone end up finding out what it was?
you should really stop smoking completely n have a healthy diet for avoiding further complications. a pack a day is sooo unhealthy and you dnt even know what you are doing to ur body.... this is ridiculous.
Garcia Angela Miller Anthony Harris Timothy
AMEN
This systematic review and meta-analysis identified significant associations of both circulating and dietary magnesium and risk of CVD events. Circulating magnesium (per 0.2-mmol/L increment) was associated with a 30% lower risk of CVD, with trends toward a lower risk of IHD and fatal IHD. Dietary magnesium was associated with a 22% lower risk of IHD and showed a nonlinear association with fatal IHD, with a 27% lower risk up to a threshold of ∼250 mg/d, compared with lower intakes. This investigation, which included a total of 313,041 individuals in whom 4106 CVD, 3215 IHD, and 1528 fatal IHD events were documented for circulating magnesium and 7889 CVD, 4319 IHD, and 1158 fatal IHD events for dietary magnesium, provides the most robust evidence to date of the associations between circulating and dietary magnesium across their usual physiologic ranges and CVD risk.
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My medical degree should have osmosis on it
This is an osomsis video , are you the same ?
Hi! We are not Osmosis but we have an active partnership with Osmosis to help promote both brands! Thanks
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