Brilliant lecture for anyone who wants to understand diabetes. Exceptionally informative for people who want to know how make better healthcare decisions about nutritional management of diabetes and metabolic syndrome. Excellently presented. Thank you.
I strongly disagree with the negative comments on this video. The information provided is as accurate as current science allows. Just because there are a bunch of quacks on the Internet who claim otherwise, doesn't make this information incorrect.
Nice video, I use Libre and Blucon and love them. The alarms are nice and are loud enough to wake me up. You can adjust your BG ranges to whatever you want. The best part of blucon is I can calibrate it for higher accuracy!
it' is not outdated but an incomplete because the topic is very complex ... to me it was like looking at one side of the coin but not the other side of coin because the metabolism burns both glucose and ketones/fats. The coverage for glucose is very good but a Type 2 Diabetic needs fats/ketone to be covered as well.
vertigoz The lipid explanation rings true! Excess lipids interfere with the insulin receptors, Blood Glucose increases, Insulin levels increase attempting to cram more glucose into cells with clogged receptor sites , so then glucose is converted to triglycerides and LDL by the liver as the kidneys try to reduce BG thru excretion, cardiovascular inflammation increases, neural and renal inflammation increase and so Metabolic Syndrome continues (one thing leads to another). The ironic thing is that people believe the dyslipedemia stems from fat intake when the carbohydrate intake is driving the bus. The internal organs are generating the lipids far more than exogenous sources I’m told. How ironic it is that a high fat very low carbohydrate diet reduces blood lipids (my labs confirm this). I’m just beginning to understand the metabolic pathways, a complicated but fascinating endeavor. The type of fat seems to be a factor. VAT (Visceral Adipose Tissue, Brown Fat, Organ Fat, etc) seem to be the culprit contributing greatly to Insulin Resistance. White Fat (Intramuscular, Subcutaneous, etc Fat) is reserved for general metabolic energy while the mitochondria-rich VAT is reserved for heat generation to maintain our 98.6 degree core temperature. Reduce VAT and Insulin Resistance decreases. (very interesting stuff)
@@vincentstouter449 Very well explained! Thanks! check out low carb down under for more info. They explain all the pathways and physiological changes, if you still need more info that is! :-)
One reason why exercise seems to help with diabetes is that it uses energy. Insulin is also an energy storing hormone. So, it seems that fasting would help diabetes type 2 and insulin in using energy so the body becomes more insulin sensitive. One problem with fasting is that it can be done wrong. One's body should be fat adapted first to avoid some issues with fasting. Then, one can fast to have lower insulin levels, while keeping fasting more sustainable and safer. Then, the body can use its stored energy and become more insulin sensitive.
Thank you for this lecture. This lecture is a comprehensive look at glycolysis/burning of glucose and how glucose management impacts the metabolism for those with Diabetes and High blood pressure. This lecture only lightly touches on the impacts of fats and the metabolism's burning of fats/ketones/triglycerides - aka ketosis. The Metabolism of fats is an important dietary component to a lifestyle intervention for diabetes. Many diabetics and those who are overweight/obese store their excess glucose as fats aka lipids in adipose/fat cells. When the metabolism is dominated by high levels of insulin - the insulin tells adipose tissue to store excess glucose as lipids/fats. There are two types of adipose tissue ... subcutaneous which is located under the skin and along side the muscle/joint to protect them from impact AND visceral which located around the vital organs to protect them from impact. Over active Subcutaneous fat won't kill you but when overactive Visceral fat gets too big, these fat cells can crush the vital organs and kill you. Non Alcohol Fatty Liver Disease (NAFLD) is an example of Visceral Fat around the liver that can kill a person. The medical solution to NAFLD is a dietary one which restricts all possible carbohydrate sources including protein. Just high dietary intake of glucose may not be enough to alter the insulin levels. High Cortisol levels will trigger hight Insulin level - under high stress cortisol levels are increase to burn glucose so the body can act quickly. Glucose burns quickly but Ketones/fats burn slowly ... burning ketones when you sleep is not a problem but the burning ketones when you are awake requires a physically fit body because burning fats produce energy more slowly. NAFLD is one of the possible common negative outcomes of Type 2 Diabetes along with renal failure. To learn about how to lose excess weight which is stored as fat - a lecture needs to be focused on ketosis and the burning of fats via getting the adipose tissue to excrete lipids to burn as fat is needed. it's complicated - but in order for the metabolism to burn fats - insulin levels must be low so ketosis can occur... the easiest way to get there not to eat 4 hours before you sleep AND to sleep for longer than 8 hours . .Note that the body comes normally with a fix number of adipose cells and depending on your fat storage needs ... adipose cells increase and decrease in size to hold lipids/fats. Each adipose cell has a maximum capacity to store lipids - there is an upper limit to how much lipid each fat cell can hold. Each body has max number of fat cells before it cannot hold anymore fat/lipids - which is genetically - epigenetically determined at birth (see the WW2 Dutch Famine Study). This is really a complex subject ....which is why people have problems with it.
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Brilliant lecture for anyone who wants to understand diabetes. Exceptionally informative for people who want to know how make better healthcare decisions about nutritional management of diabetes and metabolic syndrome. Excellently presented. Thank you.
I strongly disagree with the negative comments on this video. The information provided is as accurate as current science allows. Just because there are a bunch of quacks on the Internet who claim otherwise, doesn't make this information incorrect.
Nice video, I use Libre and Blucon and love them. The alarms are nice and are loud enough to wake me up. You can adjust your BG ranges to whatever you want. The best part of blucon is I can calibrate it for higher accuracy!
Nice outdated info, someday maybe they can get beyond research paid for by C&H sugar. Please look up how insulin, and insulin resistance work.
it' is not outdated but an incomplete because the topic is very complex ... to me it was like looking at one side of the coin but not the other side of coin because the metabolism burns both glucose and ketones/fats. The coverage for glucose is very good but a Type 2 Diabetic needs fats/ketone to be covered as well.
What actually fat does bad regarding diabetes?
When in conjunction with sugars, fat doesn't even spike insulin... again, how's fat bad for diabetes?
@@azlanfoodscapes
If fat doesn't trugger insulin how on Earth do you get insulin resistance? It has mainly to do with logic
@@azlanfoodscapes
th-cam.com/video/z33CGmmD30g/w-d-xo.html
vertigoz
The lipid explanation rings true! Excess lipids interfere with the insulin receptors, Blood Glucose increases, Insulin levels increase attempting to cram more glucose into cells with clogged receptor sites , so then glucose is converted to triglycerides and LDL by the liver as the kidneys try to reduce BG thru excretion, cardiovascular inflammation increases, neural and renal inflammation increase and so Metabolic Syndrome continues (one thing leads to another).
The ironic thing is that people believe the dyslipedemia stems from fat intake when the carbohydrate intake is driving the bus. The internal organs are generating the lipids far more than exogenous sources I’m told. How ironic it is that a high fat very low carbohydrate diet reduces blood lipids (my labs confirm this). I’m just beginning to understand the metabolic pathways, a complicated but fascinating endeavor.
The type of fat seems to be a factor. VAT (Visceral Adipose Tissue, Brown Fat, Organ Fat, etc) seem to be the culprit contributing greatly to Insulin Resistance. White Fat (Intramuscular, Subcutaneous, etc Fat) is reserved for general metabolic energy while the mitochondria-rich VAT is reserved for heat generation to maintain our 98.6 degree core temperature. Reduce VAT and Insulin Resistance decreases. (very interesting stuff)
@@vincentstouter449 Very well explained! Thanks! check out low carb down under for more info. They explain all the pathways and physiological changes, if you still need more info that is! :-)
Mam u are an disguise angel ..pls can u guide with diet for Indian vegetarian exactly for 76kg with chronic body pain
One reason why exercise seems to help with diabetes is that it uses energy. Insulin is also an energy storing hormone.
So, it seems that fasting would help diabetes type 2 and insulin in using energy so the body becomes more insulin sensitive.
One problem with fasting is that it can be done wrong. One's body should be fat adapted first to avoid some issues with fasting. Then, one can fast to have lower insulin levels, while keeping fasting more sustainable and safer. Then, the body can use its stored energy and become more insulin sensitive.
The restaurants aday, need to cook food relative to the needs of people, and not merely for profit. May ther be more vegan and low carb food.
This video needs to be amended. Very bad advice here. Add a warning that this advice is dated.
Thank you for this lecture. This lecture is a comprehensive look at glycolysis/burning of glucose and how glucose management impacts the metabolism for those with Diabetes and High blood pressure. This lecture only lightly touches on the impacts of fats and the metabolism's burning of fats/ketones/triglycerides - aka ketosis. The Metabolism of fats is an important dietary component to a lifestyle intervention for diabetes. Many diabetics and those who are overweight/obese store their excess glucose as fats aka lipids in adipose/fat cells. When the metabolism is dominated by high levels of insulin - the insulin tells adipose tissue to store excess glucose as lipids/fats. There are two types of adipose tissue ... subcutaneous which is located under the skin and along side the muscle/joint to protect them from impact AND visceral which located around the vital organs to protect them from impact. Over active Subcutaneous fat won't kill you but when overactive Visceral fat gets too big, these fat cells can crush the vital organs and kill you. Non Alcohol Fatty Liver Disease (NAFLD) is an example of Visceral Fat around the liver that can kill a person. The medical solution to NAFLD is a dietary one which restricts all possible carbohydrate sources including protein. Just high dietary intake of glucose may not be enough to alter the insulin levels. High Cortisol levels will trigger hight Insulin level - under high stress cortisol levels are increase to burn glucose so the body can act quickly. Glucose burns quickly but Ketones/fats burn slowly ... burning ketones when you sleep is not a problem but the burning ketones when you are awake requires a physically fit body because burning fats produce energy more slowly. NAFLD is one of the possible common negative outcomes of Type 2 Diabetes along with renal failure. To learn about how to lose excess weight which is stored as fat - a lecture needs to be focused on ketosis and the burning of fats via getting the adipose tissue to excrete lipids to burn as fat is needed. it's complicated - but in order for the metabolism to burn fats - insulin levels must be low so ketosis can occur... the easiest way to get there not to eat 4 hours before you sleep AND to sleep for longer than 8 hours . .Note that the body comes normally with a fix number of adipose cells and depending on your fat storage needs ... adipose cells increase and decrease in size to hold lipids/fats. Each adipose cell has a maximum capacity to store lipids - there is an upper limit to how much lipid each fat cell can hold. Each body has max number of fat cells before it cannot hold anymore fat/lipids - which is genetically - epigenetically determined at birth (see the WW2 Dutch Famine Study). This is really a complex subject ....which is why people have problems with it.
Thank you so much for your presentation! Appreciate it a bunch.
Very informative
This is incorrect
How so?
Very unprofessional and disappointing...
How so?