Disagree w/ the point at ~11min. Ligaments and Tendons adapt and get stronger w/ load regardless of age. Case in point Michael Johnson ran a 10.09s 100m in 94' (age 27) , a 19.32s 200m in 96' (age 29) and, a 43.18s 400m in 96' (age 32). Given sprinting success is multi-variable, these feats wouldn't be possible if tendons stopped adapting/growing/remodelling etc after adolescence (just seems non-sensical).
Degenerative but not painful > probably bc these cohorts aren't profoundly active (oldies that waddle around not much else). Active cohorts (play sport) with reactive and degenerative changes will have pain from experience. I think the pain is driven by the structural changes including angiogenesis develivering more blood and PG and Glycosaminoglycans causing osmosis to the area newly infiltration by nerves being innervated > pain. There may also be something to do w/ degeneration of extracellular matrix gel not allowing smooth gliding b/w fascicles etc
If one looked a at degenerative tendon image and the client was a very poor mover and inattentive (but asymptomatic) I reckon you could money on that person incurring a rupture at some point
Disagree w/ the point at ~11min. Ligaments and Tendons adapt and get stronger w/ load regardless of age. Case in point Michael Johnson ran a 10.09s 100m in 94' (age 27) , a 19.32s 200m in 96' (age 29) and, a 43.18s 400m in 96' (age 32). Given sprinting success is multi-variable, these feats wouldn't be possible if tendons stopped adapting/growing/remodelling etc after adolescence (just seems non-sensical).
Degenerative but not painful > probably bc these cohorts aren't profoundly active (oldies that waddle around not much else). Active cohorts (play sport) with reactive and degenerative changes will have pain from experience. I think the pain is driven by the structural changes including angiogenesis develivering more blood and PG and Glycosaminoglycans causing osmosis to the area newly infiltration by nerves being innervated > pain. There may also be something to do w/ degeneration of extracellular matrix gel not allowing smooth gliding b/w fascicles etc
If one looked a at degenerative tendon image and the client was a very poor mover and inattentive (but asymptomatic) I reckon you could money on that person incurring a rupture at some point