Hi medicosis, can you please make a video on brodmann areas🥺 you’re my hope now. I know, you will do a lot of research and put so much efforts for that. And i want you to know that I appreciate you 🤎
best of the best as always. I would be so happy if you could make videos about disc herniation, degenerative spine...compression fracture ...all these topics related to back pain. They r hard and there's no much explanation on TH-cam about them : (( 100% sure your explanation would take us to the next level!!
Well put video. I wanted to ask for an explanation of hyperdymamic circulation until i saw the last slide :). One last thing i find it weird and fascinating at the same time is the activation of osteoclast requires permission from the bone building units (osteoblast), what a weird and complex human composition.
Since it is preSYNCOPE, the blood will shunt from low pressure artery into high pressure venous . Hence, the blood accumulate on venous side, the overload on right atrium will damage the tricuspid valve leading to regurgitation on neck venous and inferior venocava . The heart will compensate hence supraventricular tachycardia which later will end up A.fib then sudden death!
Why not vasoconstriction because of elevated norepinephrin and KAch domination? Or is it a complication of hepatopulmonary syndrome with anastamosis and varicose in abdomen and neck ?
Another note, venous return to the left side will lead to Virchows and chylothorax lymphadenopathy. Or it will lead to thorasic duct fistula which leads to infection on lung or breast tissues .
The fistula, THATS WHY, which leads to decreased mesenteric perfusion to the gut and venous accumulation on hepatosplenomegaly which leads to ascites which leads to peritonitis and mesenteric lymphadenopathy and maybe an increase chance to a septic shock .
I see HAT size due to Ca++ uptake and cerebral edema due to VD. Also, Ca++ will conjugate with po-3 onto the bone and the muscle of scapula leading to myoalgia and kidney stone . So, optimizing vit D3 & bicarbonate will ameliorate this case.
this is not good idea that you put limit on videos i have to just watcg one video and you put that video in oart 2 antibiotic system and you thatfor one videos someine is going to give you money or pay
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Hi medicosis, can you please make a video on brodmann areas🥺 you’re my hope now. I know, you will do a lot of research and put so much efforts for that. And i want you to know that I appreciate you 🤎
Thank you!
best of the best as always. I would be so happy if you could make videos about disc herniation, degenerative spine...compression fracture ...all these topics related to back pain. They r hard and there's no much explanation on TH-cam about them : (( 100% sure your explanation would take us to the next level!!
Thank you 🙏
Well put video. I wanted to ask for an explanation of hyperdymamic circulation until i saw the last slide :). One last thing i find it weird and fascinating at the same time is the activation of osteoclast requires permission from the bone building units (osteoblast), what a weird and complex human composition.
Yup 👍
I agree with you!
To say that the human body is complex is an understatement!
Great stuff, commendable work
Thank you 🙏
Thank you sir too good🥺
Most welcome 🙏
Great video. I'd like to suggest "Pensioner" as another P mnemonic to indicate elderly british paget patients
Thank you!
youre amazing making learning is fun process
Thank you 😊
It was amazing 😊
thank you sir
i went down a rabbit hole and it lead me to pagets disease 😂 and nw i understand bones better
My pleasure 😇
Great work 👍🏻
Thanks brother for such a nice explanation❤
Thank you!
Great 😮
Thank you so much!
you are the best
Thank you so much, Heba!
Please help me by sharing my videos with others!
Please do on cases of Internal medicine and physical examination
Thank you for letting me know!
Great 😃
Thank you so much!
Since it is preSYNCOPE, the blood will shunt from low pressure artery into high pressure venous . Hence, the blood accumulate on venous side, the overload on right atrium will damage the tricuspid valve leading to regurgitation on neck venous and inferior venocava . The heart will compensate hence supraventricular tachycardia which later will end up A.fib then sudden death!
Let alone, the transudate edema on the extremities.
Why not vasoconstriction because of elevated norepinephrin and KAch domination? Or is it a complication of hepatopulmonary syndrome with anastamosis and varicose in abdomen and neck ?
Another note, venous return to the left side will lead to Virchows and chylothorax lymphadenopathy. Or it will lead to thorasic duct fistula which leads to infection on lung or breast tissues .
The fistula, THATS WHY, which leads to decreased mesenteric perfusion to the gut and venous accumulation on hepatosplenomegaly which leads to ascites which leads to peritonitis and mesenteric lymphadenopathy and maybe an increase chance to a septic shock .
I see HAT size due to Ca++ uptake and cerebral edema due to VD. Also, Ca++ will conjugate with po-3 onto the bone and the muscle of scapula leading to myoalgia and kidney stone . So, optimizing vit D3 & bicarbonate will ameliorate this case.
To add, since somatostatin stops, the IgF will increase and lead to acromegaly.
hey bro though it's gonna be a bad request but can u unlock your private videos for free?😌🤗 it'll be the best gift for medical students
First one
Thank you 🙏
this is not good idea that you put limit on videos i have to just watcg one video and you put that video in oart 2 antibiotic system and you thatfor one videos someine is going to give you money or pay
Smh
Yo! Lol
:)