I lost my father last month due to GULLIEN BARRIE syndrome..A neurologist from vishakapatnam thought its BELLS PALSY and without any second thought or second opinion he gave treatment for BELLS PALSY..My father's body got completely paralysed day by day and then we shifted him to Hyderabad..Even after ivig injection treatment also we lost him due to cardiac arrest finally..HE is 63 year old...
There’s not at all explanation for the negative history and guys the reality is ; gbs case is a horror final university practical for all the people in top medical colleges , so I sincerely request admin to conduct another brief session on gbs case with proper explanation on negative history and examination
Painful buckling ligaments Painless buckling muscle - quadriceps Normal abduct and hip flex to climb stairs Hip flex weak - cannot do quick alternate climbing - needed hand to lift thigh Hip-Dip - abductor weak Holding side rail Swing truck - glut maximus
Knee reached and upper limb involved - length dependent In this case Lmn prox motor, distal sensory -motor radiculopathy and sensory neuropathy Or radiculoneuropathy large myelinated Motor And proprioceptive Prox roots are more myelinated in radiculopathy Sensory uniform
Vasculitis Pain temp and autonomic Toxic Pain temp but spare autonomic Ahc - no sensory; purely motor Bulbar and resp can be present Muscle pain Column pattern (short column muscles are severely involved) Long column muscles Quadriceps l1 l2 l3 Short column foot muscle s2
1) Peripheral nerve -distal symmetrical mono neuritis - Distal asymmetrical; has tinel sign 2) Root - shock like; ppt factor weight lifting; relieving sign Better with lying down In sensory dermatome 3) plexus - Radiating multiple Nerve areas 4) Tract Pcolumn spindle afferent Deep boring paresthesia as if flesh is being pulled off bone Neck flex - lhermite Girdle -band like Spinothalamic Lightening pain - visceral crisis (dd aorta dissection or abdomen performation) Non length dep glove and stocking with preserved reflex Spinothalamic - superficial pain or temp
ABSOLUTELY YOU ARE GEM OF NEUROLOGY MAM, REALLY BLESSED TO HERE YOU 🙏🙏🙏
Very compitent madam full command over the subject hats off from pakistan plz take max benefit u are lucky such a wonderful teacher
Madam you are one of the best teacher till now whom I have seen..
Very informative Class. Clinical knowledge of mam is incomparable.
Thank you so much madam for this crisp and extremely short and informative discussion...you are awesome madam ... feeling blessed ❤❤🙏🙏
extremely knowledgeable Professor
She is an excellent teacher!!
Very informative discussion. Long live mam.
I lost my father last month due to GULLIEN BARRIE syndrome..A neurologist from vishakapatnam thought its BELLS PALSY and without any second thought or second opinion he gave treatment for BELLS PALSY..My father's body got completely paralysed day by day and then we shifted him to Hyderabad..Even after ivig injection treatment also we lost him due to cardiac arrest finally..HE is 63 year old...
Thanks for these awesome discussions
Thankyou so much for these lectures ....
🙏real academic feast....
There’s not at all explanation for the negative history and guys the reality is ; gbs case is a horror final university practical for all the people in top medical colleges , so I sincerely request admin to conduct another brief session on gbs case with proper explanation on negative history and examination
Excellent teacher. Command on subject...
Fan of this ma'am!🥺
Nice explanation..thank you mam
Highly inspiring lecture
Very useful mam.
the god of neurology
Superb mam thanks a lot 🎉
Great teacher, love from 🇵🇰
Aren't the reflexes preserved in classical glove and stocking pattern as well, where only small fibers are affected?
Ya isn’t it?
Glove and stocking involvement is synonymous with length dependent neuropathy due to typically involvement of large fibres
so diff things madam is saying
Great discussion...
Your fan ma'am
madam you are great
Thank you ma'am
Normal pelvis down: spine up
If Hip extensor weak
Use spine extensor to lift leg
Trunk bend: hip up
Locking knee - tripod sign
Then gower sign
Painful buckling ligaments
Painless buckling muscle - quadriceps
Normal abduct and hip flex to climb stairs
Hip flex weak - cannot do quick alternate climbing - needed hand to lift thigh
Hip-Dip - abductor weak
Holding side rail Swing truck - glut maximus
Knee reached and upper limb involved - length dependent
In this case
Lmn prox motor, distal sensory -motor radiculopathy and sensory neuropathy
Or radiculoneuropathy
large myelinated
Motor
And proprioceptive
Prox roots are more myelinated in radiculopathy
Sensory uniform
Vasculitis
Pain temp and autonomic
Toxic
Pain temp but spare autonomic
Ahc - no sensory; purely motor
Bulbar and resp can be present
Muscle pain
Column pattern (short column muscles are severely involved)
Long column muscles Quadriceps l1 l2 l3
Short column
foot muscle s2
1) Peripheral nerve -distal symmetrical
mono neuritis - Distal asymmetrical; has tinel sign
2) Root - shock like; ppt factor weight lifting; relieving sign
Better with lying down
In sensory dermatome
3) plexus -
Radiating multiple Nerve areas
4) Tract
Pcolumn spindle afferent
Deep boring paresthesia as if flesh is being pulled off bone
Neck flex - lhermite
Girdle -band like
Spinothalamic
Lightening pain - visceral crisis (dd aorta dissection or abdomen performation)
Non length dep glove and stocking with preserved reflex
Spinothalamic - superficial pain or temp
Myeloradiculoneuropathy
Sjogren
B12(sacd)
Toxic myelopathy
Distal asym sen motor
Plex and radiculoneuropathy
Why Fasciculations seen in AHC and not in muscle pathology
Why distal muscles involved in AHC and more proximal muscle involvement in muscle pathology
Thank you soo much
Thank you so much! 26/10/2020
Thank you
Thanks mam
very informative
Pdf please
367 Zboncak Ranch
Could have been better
Discussion getting deviated too much
Thank you madam🙏🏻
Thank you
Pdf please sir