Thank you so so much!! especially for the last part. I literally searched the whole internet trying to figure out why patients will be in a flexed position. A life saver!!!!
Thank you so much for the explanation. Studied for 4 years in med school and now only I can fully understand the pathophysiology of spasticity in UMN lesions
Thank you so much! My course lectures didn't explain it as well as you did, and also giving the explanation of the clinical presentation really helped!
Heard some "M'kay" here and there, which remind me of Mr. Mackey from South Park :) Jokes apart, wonderful explanation. You really have the gift of teaching!
Thank you for this, but could you explain why streaching the spastic muscle, relaxest both flexors and extensors? im just a student, ive never worked with a spastic pacient, but im very interested!
Role of gamma motor neurone in development of spasticity is not explained in this video. Other wise the video has good information for students. Thanks for your contribution.
Thank you , i fully understood your explanations on why this occurs bI'm a stroke survivor who likes answer but can i re wire this within my brain i can move my arm and get it to relax until i move or stand up then bends into the poistin within your picture :
There are some debate on wether spasticity is "just" the change of balance between inhibitory and exitatory stimulus. It doesn't really explain the different types og hypertonia - rigidity or spasticity. Why is it, that rigidity does not show increased reflexactivty
Sir do u mean Lower motor neuron is relatively controlled by inhibitory neurons than excitatory..so removal of inhibitory causes excitation..!?plz explain..
Are you referring to 'clasp knife' rigidity, where resistance suddenly 'gives' when enough force is applied? This is caused by Golgi tendon organs, which detect high levels of tension in tendons. They are part of a protective reflex (ever involuntarily dropped a weight that is too heavy for you?). Golgis are connected to inhibitory interneurones in the cord which inhibit motor neurones and cause the give. Hope that helps
I think CST neurons stimulate the inhibitory interneuron that connects to the alpha motor neuron (in blue).... Thereby inhibiting/controlling muscle contraction.
in the UE the flexors are stronger and win out over the extensors. this isn’t very scientific. how would you then explain decerebrate? really need to back up your thoughts with some science references, otherwise this really is just conjecture.
Thank you so so much!! especially for the last part. I literally searched the whole internet trying to figure out why patients will be in a flexed position. A life saver!!!!
Me too
Just a thought, why doesnt the same thing occur with rigidity? Is the agonist and antagonist affected unequally then?
Took the brakes off my learning curve, thanks, man.
Never made sense to me until now, thanks so much!
Thank you so much for the explanation. Studied for 4 years in med school and now only I can fully understand the pathophysiology of spasticity in UMN lesions
Amazing explanation ❤❤❤
Thank you so much! My course lectures didn't explain it as well as you did, and also giving the explanation of the clinical presentation really helped!
Great Explaination. Thank You !!!!
Excellent lecture. Perfectly explained.
Great video, well done.
Thank you so much Prof ... you made things super easy
You cannot explain this any better - it is perfect - clearly the Gold standard - Thank You sir -
gold standard indeed
Thanku 😊clearly explained
Awesomely explained!!!!
Wow, really good. Saudations from Brazil!
freaking brilliant presentation.
Well Done! So clear explaination!
Great explanation ✌🏼
Help me a lot! Thank you!!!
Heard some "M'kay" here and there, which remind me of Mr. Mackey from South Park :)
Jokes apart, wonderful explanation. You really have the gift of teaching!
Best explanation
awesome! thank you
Thaaaanks..easy and informative.
Learn a lot. Thank you!
Thank you, this was really clear! :)
Great vid
Very good video
Thank you! ☺️
thanks a lot Sir!!
Thank you so much 🌻
THANK YOU!
Thank you so much sir.
Thank you so much sir this is really helpful
Thank you
thank you!
Thank you sir!!
Legend
Thanks a lot 👍✅
Thanks a lot Sir
Thanks
Thank you for this, but could you explain why streaching the spastic muscle, relaxest both flexors and extensors? im just a student, ive never worked with a spastic pacient, but im very interested!
Role of gamma motor neurone in development of spasticity is not explained in this video.
Other wise the video has good information for students.
Thanks for your contribution.
Would you know why touch helps calm spasticity? Like when touching the fingers with the other hand, the fingers are easier to open
❤️
Hi Steve, do you know if there is a cure to hypertonia, or at least an effective treatment?
Thank you , i fully understood your explanations on why this occurs bI'm a stroke survivor who likes answer but can i re wire this within my brain i can move my arm and get it to relax until i move or stand up then bends into the poistin within your picture :
There are some debate on wether spasticity is "just" the change of balance between inhibitory and exitatory stimulus. It doesn't really explain the different types og hypertonia - rigidity or spasticity. Why is it, that rigidity does not show increased reflexactivty
Can the damage heal either on it's own or with some help?
Sir please make one video on spasticity vs rigidity and also types of rigidity,,,clasp knife ,lead pipe,cog wheel with their mechanisms???
so that's why my left hand can't relax at times (I have cp)
Sir do u mean Lower motor neuron is relatively controlled by inhibitory neurons than excitatory..so removal of inhibitory causes excitation..!?plz explain..
Great. Why does the spasticity ''loose up'' at the end of motion in contrast to rigidity?
Are you referring to 'clasp knife' rigidity, where resistance suddenly 'gives' when enough force is applied? This is caused by Golgi tendon organs, which detect high levels of tension in tendons. They are part of a protective reflex (ever involuntarily dropped a weight that is too heavy for you?). Golgis are connected to inhibitory interneurones in the cord which inhibit motor neurones and cause the give. Hope that helps
CST is excitatory?
I think CST neurons stimulate the inhibitory interneuron that connects to the alpha motor neuron (in blue).... Thereby inhibiting/controlling muscle contraction.
@@che-lunhu9074 Corticospinal tract is the descending excitatory tract to the alpha motor neurons to initiate voluntary movement
What part of the brain must be damaged for this to occur??
plz reply
Hello. This is caused by damage to upper motor neurones that send their axons down to the spinal cord
How neuron is pronounced is bugging me. I never heard it pronounced quite like that before.
Ventral horn is bigger than dorsal horn correct your drawing
in the UE the flexors are stronger and win out over the extensors. this isn’t very scientific. how would you then explain decerebrate? really need to back up your thoughts with some science references, otherwise this really is just conjecture.
Thank you