Hypertension - CRASH! Medical Review Series
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- เผยแพร่เมื่อ 20 พ.ย. 2015
- (Disclaimer: The medical information contained herein is intended for physician medical licensing exam review purposes only, and are not intended for diagnosis of any illness. If you think you may be suffering from any medical condition, you should consult your physician or seek immediate medical attention.)
At 2 minutes 1 second some whispered “hey I love you” then you got confused for a second and said uhhh!!.... lol.. great lecture though.. we love you too..
Haha how embarrassing! I recorded this lecture back in 2013 so I was in internship and living at my parents’ house. That was probably my mother.
How cute! 😂😂
That's fucking cute
your video's are the BEST!!! i am so grateful for your work!
Love this. I've modified the secondary HTN differential slightly which hopefully some might find easier to memorise, whilst adding a few more to the ddx and keeping the Rule of C's!:
Causes of secondary HTN
People: Conn's, Cushing
Endo: Contraceptive, CKD, CAH, Chief cells (hyperparathyroidism)
Cardiovascular/Resp: Coarctation, Closing renal artery stenosis, Can't sleep (apnoea)
Thank you. Great lectures.
Awesomely informative and perfectly explained! Thank you so much! 😊😊 17/9/2019
really great video
Thank you :)
Vanillylmandelic Acid (VMA)
Great lecture 👌 thaaaaaank you 😍
This was nice, what I clearly wanted to understand is treatment of urgency and emergency, I have a feeling urgency bp was put on ahigh level 180/120mmHg..... Coz still the patient will present with emergency symptoms, instead of treating apatient on orals in urgency why can't we first use IVs in case urgency is severe then stabilize using orals..... Margine for urgency was put so high 180/120mmhg... Had to be alittle lowered.
I am confused about the CAH treatment with Fludrocortisone. I thought fludrocortisone was a treatment for the lack of aldosterone. Also is the CAH hormone deficient in this case 11 beta hydroxylase ?
I don't understand this either and in 11-beta-hydroxylase-def. you wouldn't have aldosterone but also not any cortisol (so you technically would need to replace both).
According to my notes from the endo rotation this disease isn't actually that bad since the mineralocorticoid receptor still responds to aldosterone precursos (even if not as good); now, there's a biphasic response in patients with 11-beta-hydroxylase-def.: as infants they are suffering from Na+ wasting and while they're getting older they can accumulate precursors which will lead to K+ wasting => maybe this buildup is responsible for a late HTN?
Do anybody know about Hybetez Remedy? Does it work? I hear many people fix their high blood pressure issue inherently with this high blood pressure remedy.
i have a question why do hypertension patient have low salt diet? in fact if the kidney regulates the concentration of sodium to be in the normal range it would not matter if the patient had too much salt because the kidneys will just regulate it
Patients with HT are required to make lifestyle modification such as reducing salt intake to reduce blood volume and in turn reduce blood pressure to a level which reduces stress on the vascular system, therefore, reducing the incidence of microvascular events such as stroke, retinopathy and cardiac complications such as left ventricular hypertrophy. Although the kidney is able to regulate the system there is an underlying pathology which requires further interventions such as reducing the dietary intake of salt, in the management of the disease.
After many years of increased salt intake, the kidneys start regulating sodium and volume, and thus blood pressure in a higher level.
15:55 what do you mean by "crash box"?
EDIT: 23:40 Cushing's disease is per definitionem only an ACTH-secr. pituitary tumor.
Do anybody know about Hybetez Remedy? Does it work? I hear many individuals fix their high blood pressure issue naturally with this high blood pressure remedy.
Casey James fake and a troll