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Class 1A:Prolong the action potential duration by slowing phase 0 depolarization and delaying repolarization (phase 3). Class 1B:Mild effect on phase 0 and shortened repolarization. Class 1C :blocking rapid sodium channels, thereby slowing down the Phase 0 Class II : suppress adrenergically mediated ectopic activity. Class III: prolongation of repolarization (phase3); AP is widened and ERP is increased. Class IV : inhibit Ca2+ mediated slow channel inward current.
thank you perfect video can someone answer this : slowing down the influx of sodium ions into cardiac muscle cells causing a decrease in the excitability of the cells it has a beta adrenergic blocker which can cause bradycardia and bronchospasm : sotalol propafenone verapamil mexitilene
I have some queries :like how class 1a prolong ERP n class 1b shortens it. 2 when does class 1b act on sodium channel in open state or inactivated state.
1a prolong ERP via the effect on K+ channel blocking property.And 1b shortens via increasing diastole and extends time for recovery(Couduction=APD+DIASTOLE).1b block inactivted Na channels. hope this will help.
Love this video? Check out our ECG/EKG course made entirely with videos like this:
Students, click here: www.alilaacademy.com/courses/ecg-ekg-for-students
Teachers, click here: www.alilaacademy.com/courses/ecg-ekg-for-teachers
Class 1A:Prolong the action potential duration by slowing phase 0 depolarization and delaying repolarization (phase 3).
Class 1B:Mild effect on phase 0 and shortened repolarization.
Class 1C :blocking rapid sodium channels, thereby slowing down the Phase 0
Class II : suppress adrenergically mediated ectopic activity.
Class III: prolongation of repolarization (phase3); AP is widened and ERP is increased.
Class IV : inhibit Ca2+ mediated slow channel inward current.
you have literally saved me through CVS. Thank you so much ma'am ♥️
This was seriously soooo helpful!!! Thank you for making this and sharing it!
Wow ... I have my book Infront of me.... literally my next 40 pages in 4 minutes
So simple and so well explained, too. Thank you!
You're very welcome!
Nice , brief and helpful
Keep up with this beautiful explanations!!
Amazing beyond belief.
Thank you so much Ma'am. I couldn't understand how conduction was slowed by class 1a aa drugs.
Thank you ma❤this was soo helpful 🎉
This was very well understood.great job
This is just amazing ........thank you so much for making this
Sup future doctors?
Hey! Graduated ?
@@raphaellapapapericleous1720 5 down, 1 left. I’m coming through God willing. You?
@@Moath1277 Wish u the best :) 3 more to go for me
@@raphaellapapapericleous1720 Thanks, I wish the best for you too. Enjoy the journey. It truly is one to enjoy 👍🏼
@@Moath1277 Thank u!!!!
Thank you ma'am ❤
Simple, and much helpful, thanks.
truly awesome video and too much helpful . pls upload some videos like this
Loved this simplified explanation of anti-arrythmic drugs as well as the fact that it is short. Thank you so much!!!!
Thank you. .. saving video as usual
Happy to help!
Really really helpful..thanks a lot
i love the work guys♥
Amazing job 👏🏻
nice summary, thank you
Thank you
thanks...simple and helpful. love it 😍😍
Please make video about Thymosins(types and their functions)..thanks
thank you perfect video can someone answer this :
slowing down the influx of sodium ions into cardiac muscle cells causing a decrease in the excitability of the cells it has a beta adrenergic blocker which can cause bradycardia and bronchospasm :
sotalol
propafenone
verapamil
mexitilene
From Ethiopia thanku
Thank you Dr
Excelent!
From Iraq , thanks ❤❤❤
Thank you soooo much🤩😇
Thanks
Thank u
thank you it's very helpful
I’m on a 1C.
Do anti-arrhythmic impact contractile tissue in other muscles?
This is amaxing thank you , you helped me ALOT
Dear Sir,
Can u please make a video on the latest treatment for B-cell acute lymphoblastic Leucemia by novartis called #kymriah ?
I have some queries :like how class 1a prolong ERP n class 1b shortens it. 2 when does class 1b act on sodium channel in open state or inactivated state.
1a prolong ERP via the effect on K+ channel blocking property.And 1b shortens via increasing diastole and extends time for recovery(Couduction=APD+DIASTOLE).1b block inactivted Na channels.
hope this will help.
Damn beautiful
please upload a video on
WPW Syndrome.
Great
How can Ic drugs have no effect on ERP if they stay attached to channels the longest?
they act only on phase 0 Na channels.no other effect(ANS).
The bestest video available on this stupid topic!
What is widening the Ap ?
Please reply
Action potential ☠️
👌
Ulcerative colitis please
quick and dirty