Awesome breakdown! I like the start of the series and to begin things off with some physiology to get everyone talking the same language to build up for future lessons. Can't wait to hear the rest. ETCo2 is a great tool and important for people to understand.
You are such a blessing. Thank you!!! I Love your passion. I was never instructed in this much depth and detail in school. I have been a therapist for 10 yrs and because of you, I can now be the therapist I always wanted to be. We need more of you!!!! Blessings
Wow, thank you! I appreciate your kind words, Tonya. I think it's awesome that after 10 years, you are still perfecting your craft. A true testament to never stop learning!!
Before I found this ETCO2 videos I was about to ask you to make a video this subject. I watched all the videos and you covered all the areas that I needed. Thank you 🙏
Thank you for these! Such a nice simple breakdown. I’m an ER nurse and these are so helpful for understand basic biological functions and why pts present to the ER the way they do!
Five months ago, You answered a question I had about ETCO@. And it meant a lot. We use Sidestream, and had problems with condensation. Believe me! Condensation getting into the module is not fun to get out. Now we're getting Mainstream ETCO2 for our Trilogy EVO ventilator. It's going to be a Capnostat Mainstream ETCO2 system, and from what I've been told, condensation effects these mainstream modules even worse than Sidestream, so I'm not sure what to expect. The Capnostat will plug into the USB port on the Trilogy EVO, via a USB adapter and It'll connect to the patient's trach tube. I'm not sure what to expect about condensation, or any of that. We're use to the sampling going from the airway, to the module, where there's a vacuum pump, and you feel a tiny bit of vibration from the pump. Now, we're going to have to learn what to expect with mainstream.
I have a question. We use a CO2 module, for a CMS 8000 patient monitor, but we're having issues with the patient's on humidity through the ventilator. What can we do to stop the humidification from getting in the sense line of the etco2 adapter, and then getting into that module? So that we can monitor CO2 continuously through the Endotracheal/Tracheostomy patient? We really need some assistance!
Thank you for these videos. I don't understand why hyperventilation is a problem. Essentially, my understanding is the cells produce the CO2 as waste, then it goes through the process you draw above and the lungs exchange the CO2 for O2. You want to get rid off all (within reason) the CO2 in your body, why would your body care if the lungs are getting rid of the CO2 very quickly?
Would you mind please explain to me the effect of C02 on blood pressure? You mention that C02 causes vasodilation, which decreases your BP as well as your CO, but I thought that Hypercapnic patients with excessive C02 causes systemic vasodilation and not vasoconstriction, and it’s the opposite in the pulmonary circulation
You said it correct. " but I thought that Hypercapnic patients with excessive C02 causes systemic vasodilation " That's true! And vice versa in pulmonary circulation.
Hello, Mohan. That's when your critical thinking RT mind goes to work. You have to figure it out. Use blood pressure to assess circulation. Use temperature to assess possible metabolic causes. Of course minute ventilation, or lung improvement or worsening to assess ventilation. If your minute ventilation is fixed and not changing, yet etco2 is, then it must be cardiac, metabolic, or a change in lung function. If that same patient is afebrile, yet hypotensive, then it's probably perfusion related. If that same patient is normotensive, yet spikes a fever, then it's probably metabolic. If that same patient is normotensive and afebrile, then it's most likely a change in lung function. Does that make sense? I hope so. Thank you for watching and commenting with your doubt.
@@RespiratoryCoach very well explained. I am an engineer. I had to learn capnography for my next project. You have explained it clearly. Thank you for your videos and keep coaching ✌️
I've personally found it to have limited utility due to dead space. So often it gets misused/ misinterpreted and just confuses a situation. I think the real future is Volumetric Capnography to monitor pulmonary dead space.
I agree, Nicholas. The fact that it gets misused/misinterpreted though is not the fault of the technology, rather the lack of understanding of the practitioners. You can't have one RT that knows how to use it effectively, and 6 others that don't. That will definitely, as you stated, confuse situations. And yes, volumetric capnography!!!!
I cordially disagree with you Farhan. Coach does a great job in teaching but also providing perspective and context to what we are learning. I have seen other great youtube videos on end-tidal but only he has elaborated on why its important to respiratory therapists.
Vet Student from South Africa in Anaesthesio --- this is by far the simplest and best explanation for on the subject..
Thank you so much!!!
This vital sign needs to be taught in nursing school. Wasn't even briefly mentioned in my program.
Awesome breakdown! I like the start of the series and to begin things off with some physiology to get everyone talking the same language to build up for future lessons. Can't wait to hear the rest. ETCo2 is a great tool and important for people to understand.
Thanks, Eddie. Always appreciate your input.
Paramedic here. This was Excellent
Hey, Christopher. All disciplines are welcome here, so welcome to the channel. Thanks for watching and commenting, my fellow provider!
Don’t forget about us in paramedic school! This is a great tool for our mobile hospital
You are such a blessing. Thank you!!! I Love your passion. I was never instructed in this much depth and detail in school. I have been a therapist for 10 yrs and because of you, I can now be the therapist I always wanted to be. We need more of you!!!! Blessings
Wow, thank you! I appreciate your kind words, Tonya. I think it's awesome that after 10 years, you are still perfecting your craft. A true testament to never stop learning!!
Before I found this ETCO2 videos I was about to ask you to make a video this subject. I watched all the videos and you covered all the areas that I needed. Thank you 🙏
Awesome! Glad you found the series helpful. Thank you so much for watching and commenting 😊
Former RT here... Let's just say "physician agrees". Great job!
Thank you for these! Such a nice simple breakdown. I’m an ER nurse and these are so helpful for understand basic biological functions and why pts present to the ER the way they do!
Five months ago, You answered a question I had about ETCO@. And it meant a lot. We use Sidestream, and had problems with condensation. Believe me! Condensation getting into the module is not fun to get out. Now we're getting Mainstream ETCO2 for our Trilogy EVO ventilator. It's going to be a Capnostat Mainstream ETCO2 system, and from what I've been told, condensation effects these mainstream modules even worse than Sidestream, so I'm not sure what to expect. The Capnostat will plug into the USB port on the Trilogy EVO, via a USB adapter and It'll connect to the patient's trach tube. I'm not sure what to expect about condensation, or any of that. We're use to the sampling going from the airway, to the module, where there's a vacuum pump, and you feel a tiny bit of vibration from the pump. Now, we're going to have to learn what to expect with mainstream.
Yes, Peter! You'll have to keep me updated on the move to mainstream monitoring. I'm curious to hear about the switch. Pros and cons to both!
may this great person live long , sir i have learned lot of things from your videos , thank you very much , iam from srilanka
So nice of you! Thank you for the kind comment and for watching from Srilanka!
All the love from Romania, coach
Thank you sir! I really appreciate you and the lectures you post. Helps me a lot. I just started my ICU orientation and these videos help.
We are using it at the hospital where I work.
Thanks for breaking everything down brother man ✊🏾, Therapist from the East coast
Hey CAnderson! What part of the East Coast? And you're very welcome. Thank you for watching and commenting!
Thank you sir, you’re a beast. I’m a new RRT and needed to brush up on some things. Much appreciated!
Love the way you breakdown the concept 👌
Thanks, Khalid. Always enjoy your comments and interaction with the channel.
This is so helpful... THank you so much
You're so welcome! Thank you for watching!!
Thank you. Test tomorrow includes ETCO2
Perfect! Go kill it! Thanks for watching, Andrew.
Love the video! Thank you for always sharing your knowledge
Thanks, Jerrick! When do you graduate my man? I've really appreciated you watching and commenting!!!
Great video, on your next video I think you should mention the normal value for End title CO2 which is 35 to 45, thank you Joe
Thanks and yes, most definitely will. You're one step ahead of me!!!
thank you sir..im from hyd...
this video is GOLD
Good video.
Thank you so much it is clear and beautiful explanation
You're very welcome, Mohamed. Thank you for watching and kindly commenting.
beautiful content
I have a question. We use a CO2 module, for a CMS 8000 patient monitor, but we're having issues with the patient's on humidity through the ventilator. What can we do to stop the humidification from getting in the sense line of the etco2 adapter, and then getting into that module? So that we can monitor CO2 continuously through the Endotracheal/Tracheostomy patient? We really need some assistance!
Hey Peter. Can you send me a picture of the sampling line you are using? Respiratorycoach@gmail.com
amazing thank you sir 👍🏻
Most welcome! Thank you for watching and commenting!
Excellent Video.
I'm not sure I understand why cardiac output decreasing decreases etco2. Wouldn't co2 build up in the blood and get higher?
Excellent coach..:)
Glad you liked it, Donna!!!
Great video! Could you explain why hypoventilation increases ETco2? Or it increases ETco2/Vt ratio ?
Thank you for these videos. I don't understand why hyperventilation is a problem. Essentially, my understanding is the cells produce the CO2 as waste, then it goes through the process you draw above and the lungs exchange the CO2 for O2. You want to get rid off all (within reason) the CO2 in your body, why would your body care if the lungs are getting rid of the CO2 very quickly?
Because it creates an imbalance of pH causing an alkalosis, which causes vasoconstriction and a left shift of the oxyhemoglobin dissociation curve.
GOLD!
Thanks for watching and kindly commenting!
Thank you
Can you discuss the hfov please?
Solid stuff! Do you think measuring etCO2 in aerobic athletes can be of value?
Thanks, Rondel. Perhaps, but I have no first hand knowledge to speak of in that arena. Thanks for watching and following on my various platforms.
RT for 7 months here. Just reviewing.
Would you mind please explain to me the effect of C02 on blood pressure? You mention that C02 causes vasodilation, which decreases your BP as well as your CO, but I thought that Hypercapnic patients with excessive C02 causes systemic vasodilation and not vasoconstriction, and it’s the opposite in the pulmonary circulation
You said it correct. " but I thought that Hypercapnic patients with excessive C02 causes systemic vasodilation " That's true! And vice versa in pulmonary circulation.
I have one doubt. When etCO2 increaes/decreases, how will you diagnose whether problem is in ventilation or metabolic or circulation ?
Hello, Mohan. That's when your critical thinking RT mind goes to work. You have to figure it out. Use blood pressure to assess circulation. Use temperature to assess possible metabolic causes. Of course minute ventilation, or lung improvement or worsening to assess ventilation. If your minute ventilation is fixed and not changing, yet etco2 is, then it must be cardiac, metabolic, or a change in lung function. If that same patient is afebrile, yet hypotensive, then it's probably perfusion related. If that same patient is normotensive, yet spikes a fever, then it's probably metabolic. If that same patient is normotensive and afebrile, then it's most likely a change in lung function. Does that make sense? I hope so. Thank you for watching and commenting with your doubt.
@@RespiratoryCoach very well explained. I am an engineer. I had to learn capnography for my next project. You have explained it clearly. Thank you for your videos and keep coaching ✌️
I believe you got CO cardiac output confused with CO2
I've personally found it to have limited utility due to dead space. So often it gets misused/ misinterpreted and just confuses a situation. I think the real future is Volumetric Capnography to monitor pulmonary dead space.
I agree, Nicholas. The fact that it gets misused/misinterpreted though is not the fault of the technology, rather the lack of understanding of the practitioners. You can't have one RT that knows how to use it effectively, and 6 others that don't. That will definitely, as you stated, confuse situations. And yes, volumetric capnography!!!!
I love your videos as usual. I feel like now you are giving way too long of an intro. Thanks for the video.
Thanks for the feedback, Farhan. And as usual, thanks for watching!!!
I cordially disagree with you Farhan. Coach does a great job in teaching but also providing perspective and context to what we are learning. I have seen other great youtube videos on end-tidal but only he has elaborated on why its important to respiratory therapists.
Thank you