One major source of OPP is missing - TCP contact from fume events in airline operation. TCP in service operation around aircraft engines is bad enough, but being exposed to TCP fumes is worse possible form.
Yes, decontamination first! You *must* ensure your patient is not continuing to absorb more poison, and ensure that you and your staff are safe *first*
I used to work in a fish plant. In this plant we soaked the shrimp in giant vats of "slurry". The bags of chemical we used to make the slurry weight 75 pounds. I was fit and strong and so I cut and poured every bag of this stuff during my shift ( more or less volunteered myself ). I wound up with all kinds of crazy symptoms that plagued me until about 6 months after I left that plant. This was almost 15 years ago. I had chronic headaches, wild heart palpitations and chest pains, pains in my kidney area's, fasciculations and probably some other stuff. I didn't know what was doing this to me perhaps a post viral syndrome, maybe C0 exposure but nobody else seamed bothered. But to this day I still have the cardboard cut off from the giant box the chemical came in since I had my suspicions that this could have been a part of it. I'll read what's on it and please let me know if any of this stuff means anything to you. Sodium tripolyphosphate, Sodium Hexameta Phosphate, Natrium Chlorid, Sodium Acid Pyro Phosphate, Natrium carbonat, Sodium Citrate.
You mention tachycardia earlier in the video under "what will I see?" slide then list bradycardia in both mnemonics due to Cholinergic toxicity, does that mean tachycardia an early/late sign of compensation/decompensation?
Sounds like Shaun Murphey voice
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Exactly
Ikr😂😂
It s her sister voice
Thank you so much.
Studying for my exams and you've really helped 🙏
Best of luck!
thank u, i just wish you'd talked about the active sites, how it works
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THIS IS VERY HELPFUL FOR MY EXAM🤗
Thanks for the presentation, brief and very detailed.
One major source of OPP is missing - TCP contact from fume events in airline operation. TCP in service operation around aircraft engines is bad enough, but being exposed to TCP fumes is worse possible form.
Good info Kermit!
Yes, decontamination first! You *must* ensure your patient is not continuing to absorb more poison, and ensure that you and your staff are safe *first*
I used to work in a fish plant. In this plant we soaked the shrimp in giant vats of "slurry". The bags of chemical we used to make the slurry weight 75 pounds. I was fit and strong and so I cut and poured every bag of this stuff during my shift ( more or less volunteered myself ). I wound up with all kinds of crazy symptoms that plagued me until about 6 months after I left that plant. This was almost 15 years ago. I had chronic headaches, wild heart palpitations and chest pains, pains in my kidney area's, fasciculations and probably some other stuff. I didn't know what was doing this to me perhaps a post viral syndrome, maybe C0 exposure but nobody else seamed bothered. But to this day I still have the cardboard cut off from the giant box the chemical came in since I had my suspicions that this could have been a part of it. I'll read what's on it and please let me know if any of this stuff means anything to you. Sodium tripolyphosphate, Sodium Hexameta Phosphate, Natrium Chlorid, Sodium Acid Pyro Phosphate, Natrium carbonat, Sodium Citrate.
thanks, short and concise lecture
Amazing lecture
In the experiment of organophosphorus poisoning and rescue, what are our observation indexes when the rabbit is in normal, poisoning
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You mention tachycardia earlier in the video under "what will I see?" slide then list bradycardia in both mnemonics due to Cholinergic toxicity, does that mean tachycardia an early/late sign of compensation/decompensation?
Organophosphates cause bradycardia not tachycardia as they increase the amount of acetylcholine and therefore the parasympathetic tone to the SA node
Ach cause bradycardia when it act on cholinergic muscarinic receptors and tachycardia when ach act on cholinergic nicotinic receptors
Reflex tachycardia as a response to profound bradycardia
tachycardia is false......bradycardia cholinergic feature
Reflex tachycardia secondary to profound bradycardia
Nicotinic receptors -> tachycardia
Bradycardia we give etropine6mg
Carbofuran?
Helpful indeed
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Nice
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Covid?
Thank you alot sir💜
Noice lecture
Main cause of death respiratory failure...
Couldnt listen past the tachycardia part
tachycardia is a result of the stimulation of the nicotinic receptors :)
Lwelani Mushiana yes, that is right. But it is a very rare presentation. Usually stimulation of the muscarinic receptors will predominate.
Reflex tachycardia secondary to profound bradycardia
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Emergency medicine
THANK YOU SO MUCH .
THIS IS VERY HELPFUL FOR MY EXAM🤗