i see so many inter - relation with the ( Wim Hof ) breathing , you have the 3 minute Hyperventilation sequence, then final 3-4 minute in Apnea ..... keeps you young :-)
Hyperbaric oxygen therapy for Alzheimer's dementia with positron emission tomography imaging: a case report Med Gas Res. 2018 Oct-Dec; 8(4): 181-184 Abstract A 58-year-old female was diagnosed with Alzheimer’s dementia (AD) which was rapidly progressive in the 8 months prior to initiation of hyperbaric oxygen therapy (HBOT). 18Fluorodeoxyglucose (18FDG) positron emission tomography (PET) brain imaging demonstrated global and typical metabolic deficits in AD (posterior temporal-parietal watershed and cingulate areas). An 8-week course of HBOT reversed the patient’s symptomatic decline. Repeat PET imaging demonstrated a corresponding 6.5-38% regional and global increase in brain metabolism, including increased metabolism in the typical AD diagnostic areas of the brain. Continued HBOT in conjunction with standard pharmacotherapy maintained the patient’s symptomatic level of function over an ensuing 22 months. This is the first reported case of simultaneous HBOT-induced symptomatic and 18FDG PET documented improvement of brain metabolism in AD and suggests an effect on global pathology in AD.
i see so many inter - relation with the ( Wim Hof ) breathing , you have the 3 minute Hyperventilation sequence, then final 3-4 minute in Apnea ..... keeps you young :-)
I love this. Yes!
Traducciones en español amigosss gracias!!!!
this guys great
What was the oxygen content of the hyperbaric chamber when the participants took mask off? I'm confused by the method on that part.
should be normal at 21% oxygen, I believe.
Hyperbaric oxygen therapy for Alzheimer's dementia with positron emission tomography imaging: a case report
Med Gas Res. 2018 Oct-Dec; 8(4): 181-184
Abstract
A 58-year-old female was diagnosed with Alzheimer’s dementia (AD) which was rapidly progressive in the 8 months prior to initiation of hyperbaric oxygen therapy (HBOT). 18Fluorodeoxyglucose (18FDG) positron emission tomography (PET) brain imaging demonstrated global and typical metabolic deficits in AD (posterior temporal-parietal watershed and cingulate areas). An 8-week course of HBOT reversed the patient’s symptomatic decline. Repeat PET imaging demonstrated a corresponding 6.5-38% regional and global increase in brain metabolism, including increased metabolism in the typical AD diagnostic areas of the brain. Continued HBOT in conjunction with standard pharmacotherapy maintained the patient’s symptomatic level of function over an ensuing 22 months. This is the first reported case of simultaneous HBOT-induced symptomatic and 18FDG PET documented improvement of brain metabolism in AD and suggests an effect on global pathology in AD.