Newborn Indirect Hyperbilirubinemia - CRASH! Medical Review Series

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(Disclaimer: The medical information contained herein is intended for physician medical licensing exam review purposes only, and are not intended for diagnosis of any illness. If you think you may be suffering from any medical condition, you should consult your physician or seek immediate medical attention.)

[@DrMohammed-d8o]

I thinks the nomogram has to be opposite to what has shown.....red line in the safe zone and blue line in dangerous zone ....and thank you

[@uttamkumar097]

great lecture as always...thanks a lot paul

[@bowenpeyton4087]

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[@merrickorion1954]

@Bowen Peyton Yup, been watching on Flixzone for months myself :)

[@beaujude7514]

@Bowen Peyton Yup, been watching on Flixzone for months myself :)

[@tylane6192]

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[@xBassel92x]

What is the difference between Krigler-Najjar syndrome and Gilbers syndrome ? Thank you.

[@HC-jd4rc]

I know this is very late but maybe for someone with the same question :
There is are some differences in molecular biology even if they both present an impairment of UDP glucuronosyl transferase. Krigler-Najjar is the one that really have clinical significance because Gilbert presents as very mild hyperbilirubinemia.

[@ruqayahaydary]

thank you very much

[@shresthasanjog]

awsm as always...thank u Sir

[@xDomglmao]

14:35 How does the administration of Rhogam prevent the formation of anti-D-AB?

[@ConfusedRainbow1]

Rhogam binds the fetal RBCs, which are then destroyed by the spleen or macrophages. Because the RBCs are already bound, the mother doesn't form her own anti-D antibodies.

[@xDomglmao]

@@ConfusedRainbow1 Thank you very much!

[@eliasdavidreyes8611]

thanks

[@mohiuddinalfarra5440]

thank boss

[@aronlelenora9572]

❤❤❤❤

[@noreenfaisal4576]

👍🏻